While ectopic activation of mTORC1 in peripheral tissues might be achieved by administering a substantial dose of exogenous leucine, mTORC1 signaling while in the CNS continues to be refractory very likely as a result of quick turnover and buffering of leucine from the brain18,19. The discovery of Sestrin1 and 2 https://louisbzvpe.dbblog.net/1023043/a-secret-weapon-for-ckk-e12